[Bug] Added Items have wrong or missing metadata

Thanks for sharing this wonderful plugin with us.

Adding items from this plugin does not fetch the correct metadata for articles or perhaps uses some stock defaults. for example, The paper with the DOI '10.1073/pnas.211053698' was added to my library from your plugin. The plugin generated the following data:

Item Type   Preprint
Author  A. Krtolica
Author  S. Parrinello
Author  S. Lockett
Author  P. Desprez
Author  J. Campisi
Abstract    Mammalian cells can respond to damage or stress by entering a state of arrested growth and altered function termed cellular senescence. Several lines of evidence suggest that the senescence response suppresses tumorigenesis. Cellular senescence is also thought to contribute to aging, but the mechanism is not well understood. We show that senescent human fibroblasts stimulate premalignant and malignant, but not normal, epithelial cells to proliferate in culture and form tumors in mice. In culture, the growth stimulation was evident when senescent cells comprised only 10% of the fibroblast population and was equally robust whether senescence was induced by replicative exhaustion, oncogenic RAS, p14ARF, or hydrogen peroxide. Moreover, it was due at least in part to soluble and insoluble factors secreted by senescent cells. In mice, senescent, much more than presenescent, fibroblasts caused premalignant and malignant epithelial cells to form tumors. Our findings suggest that, although cellular senescence suppresses tumorigenesis early in life, it may promote cancer in aged organisms, suggesting it is an example of evolutionary antagonistic pleiotropy.
Date    2001-10-02
URL     https://www.semanticscholar.org/paper/cfe42fa7d6cd1b5cfd7c71aa2b2a07339be24158
Date Added  3/12/2024, 4:52:15 AM
Modified    3/12/2024, 5:08:31 AM

When I manually add the work with the DOI This is what I get:

Item Type   Journal Article
Author  Ana Krtolica
Author  Simona Parrinello
Author  Stephen Lockett
Author  Pierre-Yves Desprez
Author  Judith Campisi
Abstract    Mammalian cells can respond to damage or stress by entering a state of arrested growth and altered function termed cellular senescence. Several lines of evidence suggest that the senescence response suppresses tumorigenesis. Cellular senescence is also thought to contribute to aging, but the mechanism is not well understood. We show that senescent human fibroblasts stimulate premalignant and malignant, but not normal, epithelial cells to proliferate in culture and form tumors in mice. In culture, the growth stimulation was evident when senescent cells comprised only 10% of the fibroblast population and was equally robust whether senescence was induced by replicative exhaustion, oncogenic RAS, p14 ARF , or hydrogen peroxide. Moreover, it was due at least in part to soluble and insoluble factors secreted by senescent cells. In mice, senescent, much more than presenescent, fibroblasts caused premalignant and malignant epithelial cells to form tumors. Our findings suggest that, although cellular senescence suppresses tumorigenesis early in life, it may promote cancer in aged organisms, suggesting it is an example of evolutionary antagonistic pleiotropy.
Date    2001-10-09
Language    en
Short Title     Senescent fibroblasts promote epithelial cell growth and tumorigenesis
Library Catalog     DOI.org (Crossref)
URL     https://pnas.org/doi/full/10.1073/pnas.211053698
Accessed    3/12/2024, 5:02:05 AM
Extra   1229 citations (Crossref) [2024-03-12]
Volume  98
Pages   12072-12077
Publication     Proceedings of the National Academy of Sciences
DOI     [10.1073/pnas.211053698](http://doi.org/10.1073/pnas.211053698)
Issue   21
Journal Abbr    Proc. Natl. Acad. Sci. U.S.A.
ISSN    0027-8424, 1091-6490
Date Added  3/12/2024, 5:02:05 AM
Modified    3/12/2024, 5:02:13 AM

In the previous report, at least one key (namely, Extra) was generated by other plugin(s). It would be wonderful to - at least - get the same amount of info from the plugin.

AgiNetz / semantic-zotero

[Bug] Added Items have wrong or missing metadata #2